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Oncogenic YAP accelerates S-phase entry by shortening G1, leading to replication stress and sensitivity to CHK1 inhibition.
Mechanistic discrepancies between species highlights the limitations of mouse models in advancing STING inhibition drugs for various diseases.
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News-Medical.Net on MSNBreakthrough study questions longstanding STING activation approachResearchers have long focused on the STING (Stimulator of Interferon Genes) pathway as a way to harness the immune system's ...
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